Preliminary scientific studies show conolidine may possibly inhibit unique ion channels, lessening neuronal excitability and limiting ache signals. This mechanism is particularly suitable in neuropathic suffering, the place abnormal signaling will cause persistent distress. Also, conolidine appears to influence G protein-coupled receptor (GPCR) pathways integral to suffering notion. C... https://douglasb494joo2.national-wiki.com/user