Preliminary scientific tests point out conolidine may perhaps inhibit certain ion channels, minimizing neuronal excitability and limiting ache signals. This mechanism is particularly suitable in neuropathic soreness, in which abnormal signaling causes persistent discomfort. Furthermore, conolidine seems to impact G protein-coupled receptor (GPCR) pathways integral to pain perception. ... https://lechw024dad7.wikipowell.com/user