Preliminary studies indicate conolidine might inhibit distinct ion channels, lowering neuronal excitability and restricting pain signals. This mechanism is especially relevant in neuropathic pain, in which abnormal signaling causes persistent distress. In addition, conolidine seems to influence G protein-coupled receptor (GPCR) pathways integral to discomfort perception. Computational... https://stephenr570wtp1.bloggerchest.com/profile